During slow-wave sleep, there is a significant decline in cerebral metabolic rate and cerebral blood flow.
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Studies measuring cerebral metabolic rate for glucose ( CMRglc ) and cerebral blood flow ( CBF ) have indicated an indirect measurement of synaptic activity.
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Hypothermia decreases the cerebral metabolic rate for glucose and oxygen and reduces the loss of high energy phosphates during hypoxia-ischaemia and during secondary cerebral energy failure, The simultaneous increase in cytotoxic oedema and loss of cerebral cortical activity that accompanies secondary energy failure is also prevented.
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However, due to the important influences of arterial carbon dioxide levels, cerebral metabolic rate, neural activation, activity of the sympathetic nervous system, posture, as well as other physiological variables, cerebral autoregulation is often interpreted as encompassing the wider field of cerebral blood flow regulation.
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